Background Formation of complexes between transglutaminase 2 (TG2) and gluten can mechanistically explain why TG2 serves both as B-cell autoantigen and as an enzyme that creates deamidated gluten epitopes in coeliac disease (CeD). A model has been proposed where TG2 released from shed epithelial cells encounters high concentrations of dietary gluten peptides to form these TG2:gluten c... https://www.ngetikin.com/special-pick-MVP4-Moisture-Vapor-Protection-Anti-Fracture-and-Noise-Reduction-Membrane-special-save/